ALLMedicine™ IgA Nephropathy Center

Sep 17th, 2020 - IgA nephropathy (IgAN) is a common type of chronic immune-mediated kidney disease with variable risk of progression to end-stage kidney disease. Risk stratification helps clinicians weight the potential risks and benefits of immunosuppressive ther...

Sep 16th, 2020 - IgA nephropathy (IgAN) and IgA vasculitis with nephritis (IgAV-N) are related diseases. Galactose-deficient IgA1 (Gd-IgA1) plays an important role in the pathology of IgAV-N and IgAN, so we aim to compare the serum levels of Gd-IgA1 in Chinese ped...

Sep 12th, 2020 - In IgA nephropathy (IgAN), IgA immune complexes are deposited in the mesangium and drive inflammation and extracellular matrix (ECM) remodelling. The functional links between IgA deposition, inflammation, and matrix remodelling are not well charac...

Sep 11th, 2020 - IgA nephropathy (IgAN) is the most common primary glomerulonephritis worldwide and a leading cause of end stage renal disease (ESRD). In addition to classical progression factors, other atherosclerosis-related factors, including hyperuricemia (HU)...

Sep 11th, 2020 - Eighteen known susceptibility loci for IgAN account for only a small proportion of IgAN risk. Genome-wide meta-analysis was performed in 2628 patients and 11,563 controls of Chinese ancestry, and a replication analysis was conducted in 6879 patien...

Apr 20th, 2016 - Evidence-based clinical practice guidelines for IgA nephropathy 2014.|2016|Yuzawa Y,Yamamoto R,Takahashi K,Katafuchi R,Tomita M,|diagnosis,etiology,therapy,

Sep 17th, 2020 - IgA nephropathy (IgAN) is a common type of chronic immune-mediated kidney disease with variable risk of progression to end-stage kidney disease. Risk stratification helps clinicians weight the potential risks and benefits of immunosuppressive ther...

Sep 16th, 2020 - IgA nephropathy (IgAN) and IgA vasculitis with nephritis (IgAV-N) are related diseases. Galactose-deficient IgA1 (Gd-IgA1) plays an important role in the pathology of IgAV-N and IgAN, so we aim to compare the serum levels of Gd-IgA1 in Chinese ped...

Sep 12th, 2020 - In IgA nephropathy (IgAN), IgA immune complexes are deposited in the mesangium and drive inflammation and extracellular matrix (ECM) remodelling. The functional links between IgA deposition, inflammation, and matrix remodelling are not well charac...

Sep 11th, 2020 - IgA nephropathy (IgAN) is the most common primary glomerulonephritis worldwide and a leading cause of end stage renal disease (ESRD). In addition to classical progression factors, other atherosclerosis-related factors, including hyperuricemia (HU)...

Sep 11th, 2020 - Eighteen known susceptibility loci for IgAN account for only a small proportion of IgAN risk. Genome-wide meta-analysis was performed in 2628 patients and 11,563 controls of Chinese ancestry, and a replication analysis was conducted in 6879 patien...

Oct 23rd, 2018 - Chronic viral infection can be associated with a variety of autoimmune kidney diseases, according to a review published in Rheumatic Disease Clinics. London_England/Thinkstock In particular, hepatitis C virus (HCV) infection can cause several kidn.

Aug 1st, 2017 - Action Points Corticosteroid treatment in patients with IgA nephropathy was tied to an increased risk for serious adverse events. Note that most adverse events occurred during the first 3 months of treatment, and they were primarily driven by an e...

Jul 1st, 2015 - There is no known way to stop IgA deposition in the mesangium. Tonsillectomy to reduce mucosal IgA release has been suggested but is controversial.

May 5th, 2015 - Importantly, CGD patients also demonstrate immune-related inflammatory disorders, most commonly inflammatory bowel disease, IgA nephropathy, sarcoidosis, and juvenile idiopathic arthritis. 3 In addition, both CGD patients and female carriers of X-l.

Jul 17th, 2010 - Action Points Explain to interested patients that this study found no benefit from adding azathioprine to corticosteroid treatment for IgA nephropathy. Giving a low dose of the immunosuppressant azathioprine did not add any benefit to corticostero...